α-Lipoic acid: a potential regulator of copper metabolism in Alzheimer’s disease
ATP7A-dependent copper sequestration contributes to termination of β-CATENIN signaling during early adipogenesis
Looduslik toidulisand võib aidata Alzheimeri tõbe ennetada
α-lipoic acid ameliorates consequences of copper overload by up-regulating selenoproteins and decreasing redox misbalance.
Direct Competition of ATCUN Peptides with Human Serum Albumin for Copper(II) Ions Determined by LC-ICP MS
Characterization of Uranyl (UO22+) Ion Binding to Amyloid Beta (A?) Peptides: Effects on A? Structure and Aggregation
Metabolism of copper and possibilities for its regulation
Doktorant loodab vase reguleerimisega Alzheimerit ennetada
α-Lipoic acid ameliorates consequences of copper overload by upregulating selenoproteins and decreasing redox misbalance
Residue-specific binding of Ni(II) ions influences the structure and aggregation of amyloid beta (Aβ) peptides