Protein-Driven Copper Redox Regulation: Uncovering the Role of Disulphide Bonds and Allosteric Modulation
Binding of ApoE Isoforms to Aβ Peptides and Effects on Their Fibrillization
Distinct Effects of Copper Ionophores on Intracellular Copper and Cell Viability in Wild-Type and CTR1-Deficient SH-SY5Y Cells
Binding of Hg(I) and Hg(II) Ions to Amyloid-Beta (Aβ) Peptide Variants Affect Their Structure and Aggregation
α-Lipoic acid: a potential regulator of copper metabolism in Alzheimer’s disease
Looduslik toidulisand võib aidata Alzheimeri tõbe ennetada
α-Lipoic acid ameliorates consequences of copper overload by upregulating selenoproteins and decreasing redox misbalance
α-lipoic acid ameliorates consequences of copper overload by up-regulating selenoproteins and decreasing redox misbalance.
Characterization of Uranyl (UO22+) Ion Binding to Amyloid Beta (Aβ) Peptides: Effects on Aβ Structure and Aggregation
Direct Competition of ATCUN Peptides with Human Serum Albumin for Copper(II) Ions Determined by LC-ICP MS
Metabolism of copper and possibilities for its regulation
Residue-specific binding of Ni(II) ions influences the structure and aggregation of amyloid beta (Aβ) peptides
Doktorant loodab vase reguleerimisega Alzheimerit ennetada