Histone bivalency in CNS development
Mice lacking Astn2 have ASD-like behaviors and altered cerebellar circuit properties
Nicotinamide riboside first alleviates symptoms but later downregulates dopamine metabolism in proteasome inhibition mouse model of Parkinson’s disease
Histone bivalency regulates the timing of cerebellar granule cell development
Increased Physiological GDNF Levels Have No Effect on Dopamine Neuron Protection and Restoration in a Proteasome Inhibition Mouse Model of Parkinson’s Disease
In vivo modulation of endogenous gene expression via CRISPR/Cas9-mediated 3’UTR editing
Elevated endogenous GDNF induces altered dopamine signalling in mice and correlates with clinical severity in schizophrenia